Research and Markets has announced the addition of the "Leptin Receptor (OB Receptor or CD295 or LEPR) - Pipeline Review, H1 2016" report to their offering.
Leptin Receptor (OB Receptor or CD295 or LEPR) pipeline target constitutes close to 11 molecules, out of which approximately 11 molecules are developed by Companies.
Furthermore, the author says; Leptin Receptor (OB Receptor or CD295 or LEPR) Leptin receptor or LEP-R is a protein encoded by the LEPR gene. It acts as a receptor for hormone LEP/leptin. Upon binding it mediates LEP central and peripheral effects through the activation of different signaling pathways such as JAK2/STAT3 and MAPK cascade. LEP acts as an appetite-regulating factor that induces a decrease in food intake and an increase in energy consumption by inducing anorexinogenic factors and suppressing orexigenic neuropeptides also regulates bone mass and secretion of hypothalamo-pituitary-adrenal hormones. In the periphery increases basal metabolism, influences reproductive function, regulates pancreatic beta-cell function and insulin secretion, is pro-angiogenic and affects innate and adaptive immunity.
The report 'Leptin Receptor - Pipeline Review, H1 2016' outlays comprehensive information on the Leptin Receptor (OB Receptor or CD295 or LEPR) targeted therapeutics that are being developed by Companies/Universities, complete with analysis by indications, stage of development, mechanism of action (MoA), route of administration (RoA) and molecule type.
It also reviews key players involved in Leptin Receptor (OB Receptor or CD295 or LEPR) targeted therapeutics development, features dormant and discontinued projects and latest news and press releases. Currently, the molecules developed by Companies in Phase III, Preclinical and Discovery stages are 1, 8 and 2 respectively.
Key Topics Covered:
For more information about this report visit http://www.researchandmarkets.com/research/vzrmtf/leptin_receptor
Related Topics: Anti-Obesity Drugs
View source version on businesswire.com: http://www.businesswire.com/news/home/20160831005632/en/Business Wire
Last updated on: 31/08/2016
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